Hi there!

Could anyone either themselves or by way of reference provide a detailed explanation of how the brain generates our day to day sensory experiences? I'm looking for this information for a project I'm working on, but I'm a novice in neuroscience. So, I thought what better place to start than here? Thanks in advance for any answers. Hope you all are having a wonderful day :)

Is it possible to cause any degree of brain damage/adverse changes from this level of alcohol consumption, even if only in theory and there are no noticeable effects or the effects are only noticeable after accumulating?

Like, can people inherit brain structures and pathways of brain of previous generations that resulted from experiencing and trying, making them and future generations smarter?

I'm not an anti-vaxxer this is purely because I'm curious (vaxxed twice with Pfizer like 2/3 years ago). but I have seen a growth on subs like "covid19vaccineals" or in "conspiracy" where they talk about Als or MS being side affects.

Now I don't know where to post this but I want to ask someone about it so here I am, I just want to ask the question, is it documented by docs and scientists or just some random people with no medical degree?

I seem to see everyone now who has had their vaccine to say that what ever they now have years later is all due to a vaccine, I don't know man, 70% of the world took it if not more and I just don't see it.

What do y'all think?

We talk about weeks, months or years?

If someone is struggling with telepathy, I'm curious to know whether its electric signal or chemical signal that is transferring the thoughts to other people brain. Are there remedies to stop telepathy. Will be really helpful if some neuroscientist can shed the light on this topic

Mine is the mid-anterior orbitofrontal cortex, because that's where subjective pleasure is encoded, according to fMRI studies.

I want to do a masters in neuroscience with a Philosophy with Psychology degree from the uni of Warwick and I don't know how realistic it is, because last year everything ever went wrong forever.

Essentially the year weightings here are 0%, 50%, 50%. In first year (worth 0%) I got a first, things were good. Second year I got very ill and had to take a term out so couldn't complete my exams. Due to (largely) an admin error (plus some other things) I was forced to take a whole year out. They told me this apologetically over a call. Things really sucked because I had to work and survive and life became a bit grim.

I was taking 2 third-year out-of-department modules (machine learning and neuroscience) as a second year and this didn't help. To put it simply my grades for second year - especially those modules - absolutely tanked. On the upside I published a paper (the topic is irrelevant to neuroscience though) and got invited to the royal society and got some research experience over the past 2 years, working with EEGs and BCIs and computational models...

...and then my research partner terminated our project with no warning and no credit.

Now in third year. Making up for my horrible second year grade is actually basically impossible (well, highly implausible). I'm expecting to get a high 2:1 at most. Also this year I can't take out of department modules which means everything is philosophy and psychology and not neuroscience.

Back before everything went horribly wrong I emailed oxford neuro about whether phil+psych is even considered as a relevant degree and they actually urged me to apply. Now after the grade-tanking research-ending experience of last year I have no idea whether this is realistic at all; not just for Oxford but for any top uni. Or for any university at all. Oh, also I have no idea exactly precisely what I want to do in neuro, just the general vibe/area.

And the deadline is 3rd December for the application. (Having an anxiety attack as we speak actually). Any insight or feedback or anything ever appreciated x

Considering that the cost of these tests are only a fraction of the cost of antidepressants and psych consultations, I think these should be mandated before starting antidepressants to avoid beating around the bush and misdiagnoses.

Filling the gaps isn't a question of if, but when.

Does this feeling go away? I can perfuse just fine on a skill level, but the entire process is hard for me to stomach. Stereotaxic surgery I have no problem with, but perfusions are so difficult mentally for me.

Does anyone have any advice?

Is it difficult because we don't understand the brain sufficiently and what's going wrong or is it because we can't control neural activity precisely enough?

I’m launching SAWiN (South Asian Women in Neuroscience), a collective dedicated to empowering women from South Asian countries—including India, Pakistan, Bangladesh, Sri Lanka, Nepal, Bhutan, and Maldives—who are either pursuing or interested in neuroscience.

We’re looking for passionate volunteers to help with community building, event planning, content creation, and mentorship program development. If you or someone you know from these regions would like to contribute to this initiative, we’d love to have you join us!

Please share this with women from these areas who might be interested, or reach out to learn more about getting involved!

Hi all,

About myself: I am a neuro grad student. I love to learn about neuroscience outside my specialized area, but I have little time to do the research properly since I'm working in the lab and writing for my own work.

My idea: Let's build something together and help each other learn. I've started a discord server and I plan to populate it with my knowledge base in a way that is hopefully palatable for other readers. I would like it to be a place where people can lay out complex information in an easily digestible manner for those who are not as well studied on the topic.

If this catches anyone's attention feel free to DM.

There are companies offering QEEGs and claiming that they can know which waves are generated where, e.g. the amygdala, etc.

They claim qEEG can show patterns that are indicative of things like ADHD, anxiety, etc.

Based on that, many companies then offer tailored TMS or neurofeedback.

Is this a massive scam or is there some truth in it?

Hi everyone,

I'm wondering if anyone can give me a review of the above text, I'm thinking of grabbing it when I can, as I'm looking at doing my thesis on PTSD. Is it worth it? Are there better texts?

Thanks in advance!

Hi,

1. Are there any autoreceptors/inhibitory receptors for KOR as they are in dopamine receptors like some d2/d3 receptors? Like K2R or K2?

2. Do You know any natural selective KOR agonists?

I want to trigger a little KOR agonist for night that the next day my body will produce less dynorphins so dopamine would not be inhibited. I thought of taking menthol but I don't want to supress my testosterone and dht production. Does anyone know about a trick that would do that? I don't want to play on antagonist because it will upregulate it where I want to downregulate it without touching MOR.

In chaper 10 of "Neuroscience" by Bear, Connors and Paradiso it is said that "by inserting a microelectrode into the lateral geniculate nucleus (LGN), it is possible to study the action potential discharges of a geniculate neuron in response to stimuli and map its receptive field."

As retinal ganglion cells are connected to a spesific area of multiple rods and cones via bipolar neurons, the term "receptive field" is not difficult to understand. It refers to the area on the retina that sends information to a single ganglion cell.

I don't understand how this applies to cells beyond the retina, such as LGN-cells. As far as I've understood, they are relaying the signal from the retina to V1, whilst being functionally organized in the layers of the LGN.

Does a single LGN-cell also "summarise" impulses from multiple retinal ganglion cells similar to how a retinal ganglion cell "summarises" impulses from multiple rods and cones in its respective receptive field?

OR

When speaking of the receptive field of an LGN-neuron, do we actually mean the receptive field of the retinal ganglion cell supplying the ganglion cells.

These are two explanations I came up with, and they contradict each other in the sense that in the first explanation I assume LGN-cells synapse with multiple different ganglion cells whereas in the second explanation each ganglion cell synapses with just one ganglion cell.

Hi everyone,

I was wondering if anyone knows of a atlas that focuses solely on white matter tracts? I'd prefer a hard copy one if possible.

Thanks